Research on the Mineral Iodine
The following articles are presented as a reflection for
the use of ionic minerals iodine as a dietary supplement and nutritional supplement. You
will find more on iodine here. You can also
purchase this diet supplement below.
SUDDEN INFANT DEATH SYNDROME
Med Hypotheses (ENGLAND) Jun 1993, 40 (6) p364-6
Reid found selenium (Se) deficient soils were a common
factor in two populations with the highest incidence of SIDS world-wide (Indian population
of King County Washington State, USA and the population of Canterbury, New Zealand). Reid
compared a map of the selenium responsive livestock areas in New Zealand with areas of low
soil iodine (I). She found that the 1989 report of areas of
the highest SIDS incidence coincided with Se responsive livestock areas combined with low
soil iodine. Foster found the 1983-84 incidence of SIDS in
USA has the strongest positive correlation with the incidence of goitre in World War I
troops (1916). Sodium and strontium were positively related. Emery found a hypernatraemic
SIDS victim in a very cold environment. Robertson and Parker associated increased sodium
(Na) (used in Scunthorpe, England, as a water softener) with increased incidence of SIDS.
Godwin's study of White Muscle Disease in lambs describes Se protection. Skeletal muscle
mitochondria from dystrophic animals showed lowered respiratory rates with
palmitoyl-dl-carnitine and acetyl-dl-carnitine as substrate. Dystrophic organelles failed
to respond to ADP. Rognun found elevated hypoxanthine and an enhanced immune response in
most SIDS victims. A syndrome associated with potassium deficiency has been described as a
killer of healthy, young Asian men, most often during sleep. This paper describes the
interactions of sodium (Na), potassium (K), selenium (Se), and iodine
(I) to some factors affecting the utilization of oxygen and the production of energy. (19
Refs.)
GENERAL IODINE RESEARCH
Levander OA; Whanger PD
U.S. Department of Agriculture, Agricultural Research Service, Beltsville Human Nutrition
Research Center, MD 20705, USA.
J Nutr (UNITED STATES) Sep 1996, 126 (9 Suppl) p2427S-2434S
Information is presented regarding the approaches that have
been used to establish dietary recommendations for selenium and iodine.
In the case of selenium, activity of the selenoenzyme glutathione peroxidase has served as
a convenient biochemical endpoint for judging nutritional status. However, there are
differences of opinion among various nutritionists as to whether full expression of this
enzymatic activity is required for adequate nutriture, thereby resulting in differences in
dietary recommendations. Endpoints for assessing selenium overexposure are much less
satisfactory, but toxicological standards for selenium have nevertheless been established.
Thus far, no nutritionists have attempted to shift the paradigm for determining dietary
selenium recommendations away from prevention of deficiency disease to prevention of
chronic degenerative disease (e.g., cancer). In the case of iodine,
urinary excretion of the element is the most widely used endpoint for judging nutritional
status. Numerous epidemiological surveys have been conducted to determine the level of
urinary iodine excretion that is consistent with prevention
of goiter, the most common endpoint of iodine deficiency.
Because dietary iodine is essentially quantitatively excreted
in the urine, determination of the latter in goitrous areas will allow an almost direct
estimation of those intakes at risk of developing deficiency disease. Iodine toxicity is
complicated by the fact that some persons are quite tolerant to the element whereas others
are highly sensitive to it. There are relatively complete data sets concerning exposure
vs. human health effects for both selenium and iodine so that
sounder bases probably exist for their dietary recommendations than for many other trace
elements.
Selenium is an essential trace element in nutrition for the
prevention of disease in humans. Epidemiological studies indicate an association between
low nutritional selenium status and increased risks of cardiomyopathy, cardiovascular
disease, and carcinogenesis in various sites of the body. The role of selenium
supplementation in the prevention and treatment of AIDS-related pathology has been
considered. Selenoproteins discovered in mammalian cells may account for the essentiality
of selenium in the body's antioxidant defense; thyroid hormone function; immune system
function, particularly the cellular immunity; formation of sperm; and functioning of the
prostate gland. The seleno-organic compounds, primarily L-(+)-selenomethionine, generally
are recognized as safe and effective forms of selenium supplementation. The nutritionally
recommended dose of elemental selenium is estimated at 50 to 200 mg per day. There is,
however, increased discussion of a pharmacological dose of selenium, significantly higher
than the nutritional dose of the microelement, to treat active conditions. One way of
increasing the tissue levels of selenium is to combine its ingestible form with a nutrient
bioavailability enhancing compound. (87 Refs.)
The information contained
herein is for educational purposes only. It is not medical advice and is not intended to
replace the advice or attention of health care professionals. Consult your physician
before beginning or making changes in your diet, supplements or exercise program, for
diagnosis and treatment of illness and injuries, and for advice regarding medications.
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