Research on the Mineral Zinc
The following articles are presented as support for the
possible use of ionic minerals and zinc as a dietary supplement and nutritional
supplement. You will find more on zinc here. You
can also purchase this diet supplement below.
GENERAL ZINC RESEARCH
Beach, R. S., Gershwin, M. E., & Hurley, L. S. (1983) Am
J of Clin Nut, 38, 579-590.
Recent work has shown that offspring of outbred mice
deprived of adequate dietary zinc during the latter two
thirds of gestation exhibited a defective plaque-forming cell response to immunization
with heterologous erythrocytes, as well as impaired ontogenesis of serum Igm. Moreover,
such aberrant immunological measurements continued to be observed although to a lesser
degree, in F2 and F3 progeny. We now demonstrate that offspring of mice moderately
deprived of zinc (5 ppm zinc
diet) between days 7 and 20 of gestation also show an aberrant pattern of development of
serum levels of IgG2a and IgA, despite complete nutritional rehabilitation beginning at
birth. Only by 6 months of age were concentrations of these serum immunoglobulins similar
to those in offspring of control dams. In contrast, levels of IgG1 and IgG2b were within
normal ranges by 6 wk of age. Cross-fostering of zinc
deprived offspring to dams adequately nourished during pregnancy did little to ameliorate
their aberrant pattern of serum immunoglobulin development. Defective maturation of serum
IgG2a and IgA did not persist in F2 and F3 progeny. Nonetheless such 2nd and 3rd
generation offspring continued to have higher than normal perinatal mortality. The
alterations of immune ontogenesis in these mice could not be attributed to the persistence
of abnormal plasma zinc levels, as these were with normal
ranges. It would appear that zinc deficiency during gestation
may alter the basic mechanism of development of immunological competence.
Zinc/ Immunity/ Ontogeny/ Serum Immunoglobulin.
Anon. (1983) Int Clin Nut Rev, 3(1), 45.
Molybdenum deficiency may be the single greatest factor in
the causation of oesophageal cancer in Chinese men. Oesphageal cancer specimens taken from men living in high risk
areas have shown a significant reduction in molybdenum levels compared to normal
oesophageal specimens, and appears to be due to the low molybdenum (and zinc,) content of cereal grains and drinking water found in high
risk areas.
The anti-neoplastic properties of molybdenum include inhibition of the growth of Ehrlich
ascites tumours in mice and inhibition of nitrosamine induced stomach cancer of mice.
Levels of nitrosamines and their precursors were found to be high in staple foods
collected from high risk areas of China. Molybdenum is essential to a number of
oxidation-reduction enzymes of bacteria and algae which convert the nitrogen components in
the soil to ammonia and when plants are high in molybdenum they
contain less nitrates and nitrites.
Petering, H. G., Murphy, L., Stemmer, K. L., Finelli, V.
N., & Menden, E. E. (1986) Biological Trace Element Research, 9, 251+.
Weanling male rats were fed a copper deficient diet devoid
of cholesterol. The effects of varying source of carbohydrate and supplements of copper
and zinc on cardiovascular pathology and some biochemical and
physiological parameters were investigated. It was found that cardiomyopathy developed in
copper deficient groups. Sucrose, in contrast to starch or starch:lactose (1:1), caused
significant exacerbation of this situation. Increasing dietary Cu to 8 ppm prevented or
minimized the development of cardiomyopathy. Angiopathy occurred only when dietary zinc was at the lower level (20 ppm). Dietary copper supplements to
8.0 ppm did not alter this situation, but 120 ppm Zn in the drinking water did reduce the
angiopathy almost to the control level, except in the groups in which sucrose was fed.
Serum cholesterol was only elevated significantly over the control value when dietary
copper was deficient and sucrose was the carbohydrate source. The data point to
independent action of dietary copper or zinc
on the myocardium or vessels, respectively, with sucrose interacting to make copper and zinc supplements less active than when starch or starch/lactose was
fed.
Cardiomyopathy/ angiopathy/ essential metals/ dietary carbohydrate/ lipid metabolism/
copper deficiency/ cardiovascular.
The information contained
herein is for educational purposes only. It is not medical advice and is not intended to
replace the advice or attention of health care professionals. Consult your physician
before beginning or making changes in your diet, supplements or exercise program, for
diagnosis and treatment of illness and injuries, and for advice regarding medications.
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